Possible Mechanisms of Methylmercury Neurotoxicity: Correlation Between the Metabolic Effects of Methylmercury and 5-oxoprolinease Inhibitor on Both C6 Glial Cells and Pc12 - Ezzat Awad - Books - LAP LAMBERT Academic Publishing - 9783846585504 - June 15, 2012
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Ezzat Awad
Possible Mechanisms of Methylmercury Neurotoxicity: Correlation Between the Metabolic Effects of Methylmercury and 5-oxoprolinease Inhibitor on Both C6 Glial Cells and Pc12

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Different studies reported that glutathione (GSH) play a major role in modulating methylmercury (MeHg) neurotoxicity. Glutathione synthetase deficiency (GSD) and 5- Oxoprolinase deficiencies are two inborn errors of GSH metabolism; which were associated with physiological and pathological features resembling patients with Minamata disease (MD). The present study aimed to investigate whether there are similarities between the metabolic effects of MeHg and 5-oxoprolinase inhibitor (ICA) on both C6 Glial cells and PC12 neurons.

Media Books     Paperback Book   (Book with soft cover and glued back)
Released June 15, 2012
ISBN13 9783846585504
Publishers LAP LAMBERT Academic Publishing
Pages 104
Dimensions 150 × 6 × 226 mm   ·   173 g
Language German  

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