Ighd / Bik / Egfr / Spink1/ccna2-repressive Mechanism in Lung Adenocarcinoma - Jiang Minghu - Books - LAP Lambert Academic Publishing - 9783659670992 - December 30, 2014
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Jiang Minghu
Ighd / Bik / Egfr / Spink1/ccna2-repressive Mechanism in Lung Adenocarcinoma

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Different Pearson mutual-positive-correlation IGHD/BIK/EGFR/SPINK1/CCNA2-repressive molecular network was constructed from the overlapping of GRNInfer and Pearson under IGHD/BIK/EGFR/SPINK1/CCNA2 CC ?-0.25 in high lung adenocarcinoma compared with low human normal adjacent tissues, respectively. We propose IGHD inhibited DNA damage-induced inside-out cell differentiation through ENO1-SLC22A18-RECQL4-SQSTM1-PYCRL; BIK inhibiting axon guidance-induced membrane motility through BCC3-LGR4-DPY19L1-LIMK1; EGFR inhibited inflammatory immune-induced membrane development through CD180-TNFRSF17-POU2AF1; SPINK1 outside-inside-out inhibitive axon induced apoptosis through BUB1B-CCNA2-NCAPG-HIST1H2BD; CCNA2 inhibiting hypoxia, T cell and endocrine-induced cytoplasm differentiation through DPP41-DPP43-SLC2A5-TMEM63A-ENC12 in high lung adenocarcinoma based on integrative GO, KEGG, GenMAPP, BioCarta and disease databases. They are very useful to develop a new route and identify novel markers and potential drugs for prognosis and therapy for studying the pathogenesis

Media Books     Paperback Book   (Book with soft cover and glued back)
Released December 30, 2014
ISBN13 9783659670992
Publishers LAP Lambert Academic Publishing
Pages 108
Dimensions 7 × 152 × 229 mm   ·   179 g
Language German  

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